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Hi, can you please elaborate about tumor suppressor protein and oncovirus cell cycle.
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- certain tumor types secrete FSH-can you elaborate on this malignancy/tumor type? Can you please help me with this question? Thank you in advance!Hello! My question is: Discuss the evidence used to establish the causal association between virus infection and cancer. How can this knowledge be used in the diagnosis, treatment and prevention of virus-associated tumours? Thank you!!!!Write about tumor heterogenisity and progression
- Hi, can you elaborate about: 1. MyoD to be used to manipulate cancer cells at an early stage and then be used as a treatment. 2. Angiogenesis is a major function for tumor cells to grow, so if we break off the blood supply, would this result in better treatment?Describe the 4 stages of cancerShould he reconsider and try chemotherapy instead? Bruce and his parents moved to a semi-tropical region of the United States when he was about 3 years old. He loved to be outside year-round and swim, surf, snorkel, and play baseball. Bruce was fair-skinned, and in his childhood years, was sunburned quite often. In his teen years, he began using sunscreens, and although he never tanned very much, he did not have the painful sunburns of his younger years. After graduation from the local community college, Bruce wanted an outdoor job and was hired at a dive shop. He took people out to one of the local reefs to snorkel and scuba dive. He didnt give a second thought to sun exposure because he used sunscreen. His employer did not provide health insurance, so Bruce did not go for annual checkups, and tried to stay in good health. In his late 20s, Bruce was injured trying to keep a tourist from getting caught between the dive boat and the dock. He went to an internist, who treated his injury and told Bruce he was going to give him a complete physical exam. During the exam, the internist noticed a discolored patch of skin on Bruces back. She told him that she suspected Bruce had skin cancer and referred him to a dermatologist, who biopsied the patch. At a follow-up visit, Bruce was told that he had melanoma, a deadly form of skin cancer. Further testing revealed that the melanoma had spread to his liver and his lungs. The dermatologist explained that treatment options at this stage are limited. The drugs available for chemotherapy have only temporary effects, and surgery is not effective for melanoma at this stage. The dermatologist recommended that Bruce consider entering a clinical trial that was testing a DNA vaccine for melanoma treatment. These vaccines deliver DNA encoding a gene expressed by the cancer cells to the immune system. This primes the immune system to respond by producing large quantities of antibodies that destroy melanoma cells wherever they occur in the body. A clinical trial using one such DNA vaccine was being conducted at a nearby medical center, and Bruce decided to participate. At the study clinic, Bruce learned that he would be in a Phase Ill trial, comparing the DNA vaccine against the standard treatment, which is chemotherapy, and that he would be randomly assigned to receive either the DNA vaccine or the chemotherapy. He was disappointed to learn this. He thought he would be receiving the DNA vaccine.
- Retinoblastoma: The Hits Just Keep Coming 1.a There are three broad categories of cancer-related genes: proto-oncogenes, tumor suppressor genes, and DNA repair/stability genes. Distinguish between these three groups, then indicate which you think RB1 belongs to. b. In 1971, Alfred Knudson noticed that children like Kay, who have retinoblastoma in both eyes (bilateral), presented at earlier ages than those who had unilateral disease. To explain these different disease kinetics, he proposed the “two-hit hypothesis.” What does this hypothesis propose? c. Consider Dr. Whitt’s comments about the relationship between the wild-type and mutant RB1 alleles and what you now know about Knudson’s two-hit hypothesis. Do these ideas seem to confirm or contradict your earlier prediction (based on the pedigree) about whether the mutant RB1 allele is dominant or recessive to the wild-type allele? Explain. d. Do you think that Kay (and Julie) will be more likely to develop other cancers later in life?Why are histopathologists interested in the percentage of mitotic cells in a tissue biopsy? 1.High rates of cell division (i.e. a high mitotic index) are characteristic of tumours 2.Low rates of cell division (i.e. a low mitotic index) are indicative of tumours 3.Mature differentiated cells continue to go through the cell cycle 4.They are interested in looking for misshapen cells.The term ____ describes the ability of cancer cells to spread to other areas of the body. Group of answer choices malignant tumor metastasize benign
- just True or False 1- All cancer starts the same way are lose their normal ability to life.2- From example of hyperplasia is hyperplasia of uterine muscle during pregnancy .3-All cells of body have an inbuilt mechanism to deal with changes in environment are reversible and irreversible cell injury. 4- Cell injury results when cells are stressed so severely that they are no longer able to adapt or when cells are exposed to inherently damaging agents. Injury may progress through a reversible stage and culminate in reversible injury .ivision & Differentiation 6. Which of the following statements best explains why cancer is so difficult to cure? OA. Each cancer cell has the ability to control its own growth. OB. Each type of cancer is caused by one gene that continuously mutates. Oc. There are many different types of cancer, each caused by different factors. OD. Every type of cancer is different, but they use the same treatments. Reset Submit12/ cell determination is the expression of the cell's specialized role true or false
